| Vaccine breakthrough infections with SARS-CoV-2 variants E Hacisuleyman, C Hale, Y Saito, NE Blachere, M Bergh, EG Conlon, ... New England Journal of Medicine 384 (23), 2212-2218, 2021 | 739 | 2021 |
| The C9ORF72 GGGGCC expansion forms RNA G-quadruplex inclusions and sequesters hnRNP H to disrupt splicing in ALS brains EG Conlon, L Lu, A Sharma, T Yamazaki, T Tang, NA Shneider, ... elife 5, e17820, 2016 | 277 | 2016 |
| RNA-binding proteins in neurodegeneration: mechanisms in aggregate EG Conlon, JL Manley Genes & development 31 (15), 1509-1528, 2017 | 220 | 2017 |
| Unexpected similarities between C9ORF72 and sporadic forms of ALS/FTD suggest a common disease mechanism EG Conlon, D Fagegaltier, P Agius, J Davis-Porada, J Gregory, I Hubbard, ... Elife 7, e37754, 2018 | 72 | 2018 |
| Widespread intron retention impairs protein homeostasis in C9orf72 ALS brains Q Wang, EG Conlon, JL Manley, D Rio Genome Research, gr. 265298.120, 2020 | 42 | 2020 |
| C9orf72 and triplet repeat disorder RNAs: G-quadruplex formation, binding to PRC2 and implications for disease mechanisms X Wang, KJ Goodrich, EG Conlon, J Gao, AH Erbse, JL Manley, TR Cech Rna 25 (8), 935-947, 2019 | 40 | 2019 |
| Burkitt-lymphoma-related TCF3 mutations alter TCF3 alternative splicing by disrupting hnRNPH1 binding T Yamazaki, L Liu, EG Conlon, JL Manley RNA Biology, 2020 | 10 | 2020 |
| New York Genome Center ALSC, Phatnani H et al (2018) Unexpected similarities between C9ORF72 and sporadic forms of ALS/FTD suggest a common disease mechanism EG Conlon, D Fagegaltier, P Agius, J Davis-Porada, J Gregory, I Hubbard, ... Elife 7, e37754, 0 | 5 | |
| Shared Molecular Features of Inherited and Sporadic ALS/FTD EG Conlon Columbia University, 2018 | | 2018 |
| Henry Ford Health System Scholarly Common s EG Conlon, D Fagegaltier, P Agius, J Davis-Porada, J Gregory | | |
| Aparición en vacunados de infecciones con variantes del SARS-CoV-2 BSN Bergh, EG Conlon, DJ Schaefer‑Babajew, J DaSilva, F Muecksch, ... | | |
